notes-work-olearyTalk0911

(where corticospinal would be) v1 l5 projects to sup colliculus

emx2 pax6

spinal cord: diff genes express in specific subpopulations in progenitors in spinal cord progenitors, but not in cortex

bishop et al science 2000, j of neurosci 2002

lx2 something f1

pmbsf barrels "co histochemistry" p7 serotonin immunohistostaining

rorb

changing emx2 does not cause chimeric nrns

cad8 mega1

note: wang and burkhalter higher order areas get too close to S1, mb A -- suggest multimodality (which that other paper confirms anyway).

visalp, visam, visi are the ones oleary likes out of the genes he has

coup-tf1

note: things like coup-tf1 deletion seem more like tuning than basic property; are there severse deficits in phenotype? also, who do emx2- mice die?

mdga1

emx2 vis+, coup-tf1 motor-

sp8, pax 6 are s1-, motor+??

oleary's model is a total ordering of areal dominance

my alternate hypothesis why making motor areas larger hurts performance: mb 2nd-order areas get hurt